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Antihypoxants and antioxidants

Hypoxia (oxygen deficiency) is observed in a number of pathological conditions (ischemic heart disease, disorders of the brain, lungs, liver, kidneys, eyes, fetal abnormality, etc.). Often, hypoxia not only complicates the course of the disease, but also determines its outcome.
Hypoxia also develops with blood loss, shock, physical overload, etc.
For a long time, to reduce hypoxia, an increase in oxygen delivery to the body has been used. Hyperbaric oxygenation has become widespread in recent decades.
In recent years, pharmacological tools and methods that improve the body's utilization of oxygen circulating in it and reduce the oxygen demand of organs and tissues have aroused great interest. It was found that a number of drugs (see Nitrates; A b -blockers; Calcium ion antagonists; Nootropes, etc.) contribute to a more “economical” consumption of oxygen by the tissues, its better utilization and, thus, a decrease in hypoxia and an increase in the body’s resistance to oxygen failure.
For the correction of hypoxia, they began to create special means, called Kantihypoxants.
An important role in the fight against hypoxia is also played by Kantioxidants, which, as a rule, combine the antiradical effect and the effect on lipid peroxidation with antihypoxant activity (see Tocopherol acetate; Dibunol, Emoxipin, etc.).
Cytochrome C has a pronounced antihypoxic effect (see).